SARS-CoV-2, the virus responsible for COVID-19, causes widespread inflammation and injury in the lungs, giving rise to diffuse alveolar damage (DAD) ( Andrea Valeria Arrossi and Farver, 2020 Damiani et al., 2021 Borczuk et al., 2020 Li et al., 2021 Roden, 2020), featuring marked infection and viral burden leading to apoptosis of alveolar pneumocytes ( Hussman, 2020), along with pulmonary edema ( Bratic and Larsson, 2013 Carsana et al., 2020). Department of Medicine, University of California, San Diego, United States.Department of Pediatrics, University of California, San Diego, United States.Division of Infectious Diseases, Department of Medicine, University of California, San Diego, United States.Cell Applications Inc., United States.Division of Pulmonary and Critical Care, Department of Medicine, University of California, San Diego, United States.Pulmonary Critical Care Section, Veterans Affairs (VA) San Diego Healthcare System, United States.Division of Cardiology, Department of Internal Medicine, UC San Diego Medical Center, United States.Division of Cardiothoracic Surgery, University of California San Diego, United States.Department of Immunology and Microbiology, The Scripps Research Institute, United States.
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Department of Pathology, University of California San Diego, United States.Department of Computer Science and Engineering, Jacobs School of Engineering, University of California San Diego, United States.HUMANOID CoRE, University of California San Diego, United States.
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